Probing the Origin of the Toxicity of Oligomeric Aggregates of α-Synuclein with Antibodies | ACS Chemical Biology
Julius Bier Kirkegaard — Theoretical Biophysics & Data Science
A Brain-Permeable Aminosterol Regulates Cell Membranes to Mitigate the Toxicity of Diverse Pore-Forming Agents | ACS Chemical Neuroscience
Deletion of SERF2 in mice delays embryonic development and alters amyloid deposit structure in the brain | Life Science Alliance
Frontiers | Key Metabolic Pathways in MSC-Mediated Immunomodulation: Implications for the Prophylaxis and Treatment of Graft Versus Host Disease
The therapeutic potential of targeting tryptophan catabolism in cancer | British Journal of Cancer
Tobacco carcinogen induces tryptophan metabolism and immune suppression via induction of indoleamine 2,3-dioxygenase 1 | Signal Transduction and Targeted Therapy
Probing the Origin of the Toxicity of Oligomeric Aggregates of α-Synuclein with Antibodies | ACS Chemical Biology
Polyglutamine protein aggregates are dynamic | Nature Cell Biology
Ellen Nollen | VIB Conferences
Frontiers | Parasexuality of Candida Species
Tryptophan metabolism: entering the field of aging and age-related pathologies: Trends in Molecular Medicine
Speakers | ARDD 2022
Akira KITAMURA | Senior Lecturer | Ph.D. | Hokkaido University, Sapporo | Hokudai | Faculty of Advanced Life Science | Research profile
Untargeted Metabolomic Characterization of Glioblastoma Intra-Tumor Heterogeneity Using OrbiSIMS | Analytical Chemistry
Frontiers | Regulation of Age-Related Protein Toxicity
Probiotic Bacillus subtilis Protects against α-Synuclein Aggregation in C. elegans - ScienceDirect
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Delivery of Native Proteins into C. elegans Using a Transduction Protocol Based on Lipid Vesicles | Scientific Reports
MOAG-4 promotes the aggregation of α-synuclein by competing with self-protective electrostatic interactions - ScienceDirect
Aniket SHARMA | Research scholar | Masters of Biotechnology | University of Wyoming, WY | UW | Department of Animal Science | Research profile
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity: Cell
Immune regulation through tryptophan metabolism | Experimental & Molecular Medicine
Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity: Cell
Proteome-wide Changes in Protein Turnover Rates in C. elegans Models of Longevity and Age-Related Disease - ScienceDirect
MOAG-4 promotes the aggregation of α-synuclein by competing with self-protective electrostatic interactions - ScienceDirect
